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primary rabbit monoclonal antibodies against total and phosphorylated forms of p38, jnk, and erk  (Cell Signaling Technology Inc)


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    Cell Signaling Technology Inc primary rabbit monoclonal antibodies against total and phosphorylated forms of p38, jnk, and erk
    Primary Rabbit Monoclonal Antibodies Against Total And Phosphorylated Forms Of P38, Jnk, And Erk, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/primary+rabbit+antibodies+against+total+erk/pharmacological+inhibitors+of+p38+5633s/pm31456484-50-13-17
    Average 90 stars, based on 1 article reviews
    primary rabbit monoclonal antibodies against total and phosphorylated forms of p38, jnk, and erk - by Bioz Stars, 2026-09
    90/100 stars

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    Related Articles

    Control:

    Article Title: CCAT2 is an oncogenic long non-coding RNA in pancreatic ductal adenocarcinoma
    Article Snippet: .. Primary rabbit antibodies against total ERK (No. 4695), p-ERK (No. 4370), total AKT (No. 4685), p-AKT (No. 4060), KRAS (No. 3339) (Cell Signaling Technology Inc., Berkeley, CA, USA) and GAPDH (No. TA-08) (loading control) (Zhongshanjinqiao Biotech, China) were incubated at 4 °C overnight at a dilution of 1:1000, and after washed with PBST for three times, the secondary horseradish-peroxidase-labeled antibody was incubated at room temperature for 2 h at a dilution of 1:5000. ..

    Incubation:

    Article Title: CCAT2 is an oncogenic long non-coding RNA in pancreatic ductal adenocarcinoma
    Article Snippet: .. Primary rabbit antibodies against total ERK (No. 4695), p-ERK (No. 4370), total AKT (No. 4685), p-AKT (No. 4060), KRAS (No. 3339) (Cell Signaling Technology Inc., Berkeley, CA, USA) and GAPDH (No. TA-08) (loading control) (Zhongshanjinqiao Biotech, China) were incubated at 4 °C overnight at a dilution of 1:1000, and after washed with PBST for three times, the secondary horseradish-peroxidase-labeled antibody was incubated at room temperature for 2 h at a dilution of 1:5000. ..



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    primary rabbit monoclonal antibodies against total and phosphorylated forms of p38, jnk, and erk  (Cell Signaling Technology Inc)
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    Cell Signaling Technology Inc primary rabbit monoclonal antibodies against total and phosphorylated forms of p38, jnk, and erk
    Primary Rabbit Monoclonal Antibodies Against Total And Phosphorylated Forms Of P38, Jnk, And Erk, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/primary+rabbit+antibodies+against+total+erk/pharmacological+inhibitors+of+p38+5633s/pm31456484-50-13-17
    Average 90 stars, based on 1 article reviews
    primary rabbit monoclonal antibodies against total and phosphorylated forms of p38, jnk, and erk - by Bioz Stars, 2026-09
    90/100 stars
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    primary rabbit antibodies against total erk  (Cell Signaling Technology Inc)
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    Cell Signaling Technology Inc primary rabbit antibodies against total erk
    KRAS regulates CCAT2 expression via <t>MEK/ERK</t> pathway. a The expression of KRAS was compared between Scramble- and si-KRAS-treated PANC-1 cells 48 h after transfection. b CCAT2 expression in PANC-1 cells transfected with si-KRAS or Scramble. *P < 0.05. c Immunoblotting for p-ERK, <t>total</t> <t>ERK,</t> p-AKT and total AKT in si-KRAS-treated and Scramble-treated PANC-1 cells 48 h after transfection. d PANC-1 cells were co-transfected with CCAT2 promoter-reporter plasmid in combination with si-KRAS or Scramble. Luciferase activity were measured 48 h after transfection. *P < 0.05. For a and c , GAPDH was used as loading control. Image shown was representative of three independent biological repeats. For b and d , data shown were mean ± SEM of three independent biological repeats. e Activation of ERK or AKT was evaluated in PANC-1 cells treated with MEK/ERK inhibitor PD98059 or PI3K/AKT inhibitor LY294002 for 24 h. f CCAT2 expression in PANC-1 cells treated with PD98059 or LY294002 for 24 h. *P < 0.05. For e and f , DMSO-treated group was used as control. Image shown was representative of three independent biological repeats
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    primary rabbit antibodies against total erk - by Bioz Stars, 2026-09
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    primary rabbit antibodies against total and phosphorylated p38, jnk and erk  (Signalway Antibody)
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    Signalway Antibody primary rabbit antibodies against total and phosphorylated p38, jnk and erk
    KRAS regulates CCAT2 expression via <t>MEK/ERK</t> pathway. a The expression of KRAS was compared between Scramble- and si-KRAS-treated PANC-1 cells 48 h after transfection. b CCAT2 expression in PANC-1 cells transfected with si-KRAS or Scramble. *P < 0.05. c Immunoblotting for p-ERK, <t>total</t> <t>ERK,</t> p-AKT and total AKT in si-KRAS-treated and Scramble-treated PANC-1 cells 48 h after transfection. d PANC-1 cells were co-transfected with CCAT2 promoter-reporter plasmid in combination with si-KRAS or Scramble. Luciferase activity were measured 48 h after transfection. *P < 0.05. For a and c , GAPDH was used as loading control. Image shown was representative of three independent biological repeats. For b and d , data shown were mean ± SEM of three independent biological repeats. e Activation of ERK or AKT was evaluated in PANC-1 cells treated with MEK/ERK inhibitor PD98059 or PI3K/AKT inhibitor LY294002 for 24 h. f CCAT2 expression in PANC-1 cells treated with PD98059 or LY294002 for 24 h. *P < 0.05. For e and f , DMSO-treated group was used as control. Image shown was representative of three independent biological repeats
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    primary rabbit antibodies against total and phosphorylated p38, jnk and erk - by Bioz Stars, 2026-09
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    primary rabbit-polyclonal antibodies against total erk  (Cell Signaling Technology Inc)
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    Cell Signaling Technology Inc primary rabbit-polyclonal antibodies against total erk
    KRAS regulates CCAT2 expression via <t>MEK/ERK</t> pathway. a The expression of KRAS was compared between Scramble- and si-KRAS-treated PANC-1 cells 48 h after transfection. b CCAT2 expression in PANC-1 cells transfected with si-KRAS or Scramble. *P < 0.05. c Immunoblotting for p-ERK, <t>total</t> <t>ERK,</t> p-AKT and total AKT in si-KRAS-treated and Scramble-treated PANC-1 cells 48 h after transfection. d PANC-1 cells were co-transfected with CCAT2 promoter-reporter plasmid in combination with si-KRAS or Scramble. Luciferase activity were measured 48 h after transfection. *P < 0.05. For a and c , GAPDH was used as loading control. Image shown was representative of three independent biological repeats. For b and d , data shown were mean ± SEM of three independent biological repeats. e Activation of ERK or AKT was evaluated in PANC-1 cells treated with MEK/ERK inhibitor PD98059 or PI3K/AKT inhibitor LY294002 for 24 h. f CCAT2 expression in PANC-1 cells treated with PD98059 or LY294002 for 24 h. *P < 0.05. For e and f , DMSO-treated group was used as control. Image shown was representative of three independent biological repeats
    Primary Rabbit Polyclonal Antibodies Against Total Erk, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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    Image Search Results


    KRAS regulates CCAT2 expression via MEK/ERK pathway. a The expression of KRAS was compared between Scramble- and si-KRAS-treated PANC-1 cells 48 h after transfection. b CCAT2 expression in PANC-1 cells transfected with si-KRAS or Scramble. *P < 0.05. c Immunoblotting for p-ERK, total ERK, p-AKT and total AKT in si-KRAS-treated and Scramble-treated PANC-1 cells 48 h after transfection. d PANC-1 cells were co-transfected with CCAT2 promoter-reporter plasmid in combination with si-KRAS or Scramble. Luciferase activity were measured 48 h after transfection. *P < 0.05. For a and c , GAPDH was used as loading control. Image shown was representative of three independent biological repeats. For b and d , data shown were mean ± SEM of three independent biological repeats. e Activation of ERK or AKT was evaluated in PANC-1 cells treated with MEK/ERK inhibitor PD98059 or PI3K/AKT inhibitor LY294002 for 24 h. f CCAT2 expression in PANC-1 cells treated with PD98059 or LY294002 for 24 h. *P < 0.05. For e and f , DMSO-treated group was used as control. Image shown was representative of three independent biological repeats

    Journal: Biological Research

    Article Title: CCAT2 is an oncogenic long non-coding RNA in pancreatic ductal adenocarcinoma

    doi: 10.1186/s40659-017-0149-0

    Figure Lengend Snippet: KRAS regulates CCAT2 expression via MEK/ERK pathway. a The expression of KRAS was compared between Scramble- and si-KRAS-treated PANC-1 cells 48 h after transfection. b CCAT2 expression in PANC-1 cells transfected with si-KRAS or Scramble. *P < 0.05. c Immunoblotting for p-ERK, total ERK, p-AKT and total AKT in si-KRAS-treated and Scramble-treated PANC-1 cells 48 h after transfection. d PANC-1 cells were co-transfected with CCAT2 promoter-reporter plasmid in combination with si-KRAS or Scramble. Luciferase activity were measured 48 h after transfection. *P < 0.05. For a and c , GAPDH was used as loading control. Image shown was representative of three independent biological repeats. For b and d , data shown were mean ± SEM of three independent biological repeats. e Activation of ERK or AKT was evaluated in PANC-1 cells treated with MEK/ERK inhibitor PD98059 or PI3K/AKT inhibitor LY294002 for 24 h. f CCAT2 expression in PANC-1 cells treated with PD98059 or LY294002 for 24 h. *P < 0.05. For e and f , DMSO-treated group was used as control. Image shown was representative of three independent biological repeats

    Article Snippet: Primary rabbit antibodies against total ERK (No. 4695), p-ERK (No. 4370), total AKT (No. 4685), p-AKT (No. 4060), KRAS (No. 3339) (Cell Signaling Technology Inc., Berkeley, CA, USA) and GAPDH (No. TA-08) (loading control) (Zhongshanjinqiao Biotech, China) were incubated at 4 °C overnight at a dilution of 1:1000, and after washed with PBST for three times, the secondary horseradish-peroxidase-labeled antibody was incubated at room temperature for 2 h at a dilution of 1:5000.

    Techniques: Expressing, Transfection, Western Blot, Plasmid Preparation, Luciferase, Activity Assay, Control, Activation Assay